NR
Nicotinamide Riboside (Niagen / Tru Niagen)
NAD+ precursor: two enzymatic steps from NAD+ (NR → NMN → NAD+). Most-studied NAD+ precursor with multiple human RCTs showing dose-dependent elevation of blood NAD+ (22% at 100 mg, 51% at 300 mg, 142% at 1000 mg in Conze 2019). Marketed primarily as Niagen (ChromaDex) and Tru Niagen. Clear regulatory status as a dietary supplement (GRAS notification accepted by FDA, multiple NDIs accepted). Generally regarded as the safer/cleaner option vs NMN due to longer history of human dosing.
At a glance
- Half-life
- ~2.7 h Plasma NR Tmax ~2.7h; rapidly metabolized. NAD+ pool elevation persists for hours-to-days post-dose; chronic dosing produces sustained NAD+ elevation over 2 weeks.
- Routes
- Oral
- Evidence base
- 5 studies 5 human
Mechanism
Oral NR enters cells via equilibrative nucleoside transporters (ENTs). Phosphorylated to NMN by NRK1/NRK2 kinases, then converted to NAD+ by NMNAT. Bypasses some salvage pathway steps, resulting in direct NAD+ pool elevation. Like NMN, raises NAD+ for downstream sirtuin and PARP activity. Some NR is also de-glycosylated to nicotinamide before re-entering salvage.
Dosing
Any amounts shown here are reported from published studies only. 5 of 5 catalogued studies involved human subjects. COMPOUND does not publish its own dosing guidance.
Where a study reports an amount, it appears in that study's entry below, attributed to the source.
Published evidence
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Human RCT 2023
NR-SAFE: a randomized, double-blind safety trial of high dose NR in Parkinson's disease
n=20 PD patients, 4 weeks of 1500 mg BID (3000 mg/day). Safe, no moderate or severe AEs. Pronounced augmentation of NAD metabolome. NO methyl donor depletion (SAM and homocysteine unchanged). Supports dose escalation to 3 g/day.
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Human RCT 2019
Safety and Metabolism of Long-term NIAGEN (NR Chloride) Administration in Healthy Overweight Adults
n=140, 8-week dose-ranging trial (100/300/1000 mg). Dose-dependent NAD+ elevation: +22%, +51%, +142%. No adverse events differentially attributable to NR. Foundational dose-response and safety demonstration.
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Human RCT 2019
NR Augments the Aged Human Skeletal Muscle NAD+ Metabolome and Induces Anti-inflammatory Signatures
n=12 aged men (70-80 y/o), 21 days at 1000 mg/day NR vs placebo crossover. Skeletal muscle NAD+ metabolome elevated. Reduced circulating inflammatory cytokines.
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Human RCT 2018
Chronic nicotinamide riboside supplementation is well-tolerated and elevates NAD+ in healthy middle-aged and older adults
n=24, 6-week crossover RCT, 1000 mg/day NR. Elevated whole-blood NAD+ ~60%. Trend toward reduced systolic BP and arterial stiffness in subgroup with stage 1 hypertension. Foundational human trial demonstrating PD effect of chronic NR.
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Human RCT 2018
A randomized placebo-controlled clinical trial of NR in obese men: safety, insulin-sensitivity, and lipid-mobilizing effects
n=40 obese sedentary men, 12 weeks of 1000 mg BID (2g/day). NO improvement in insulin sensitivity (clamp). No change in fasting glucose, HbA1c, lipids, or ALT. Plasma TG increased modestly. NEGATIVE result for insulin sensitivity in obese men.
Reported side effects
- Generally well-tolerated
- Very common Mild · Across multiple RCTs (Martens 2018, Dollerup 2018, Conze 2019, Elhassan 2019, NR-SAFE 2023), AE rates similar to placebo. NR-SAFE confirmed safety up to 3000 mg/day x 4 weeks. No reports of flushing (unlike high-dose niacin).
- Mild GI symptoms
- Occasional Mild · Pruritus, excessive sweating, bloating, mild stool changes, acid reflux, loose stools reported in Dollerup 2018 (4 NR vs 2 placebo participants). All mild, transient.
- Modest TG elevation at high dose
- Occasional Mild · Plasma TG increased modestly in Dollerup 2018 at 2g/day. Reasonable to monitor lipids if dosing chronically at >1g/day.
Interactions
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NMN Redundant
Same NAD+ pool target. No proven additive benefit from stacking.
Storage
Capsules room temp, dry, dark. Niagen crystal form is more stable than amorphous NR. Third-party tested products preferred; generic NR products show variable purity.